Mirna.7z Direct
The dysregulation of miR-7 is a hallmark of several major pathologies:
: In Parkinson’s disease , miR-7 levels are typically decreased, leading to the toxic accumulation of α-synuclein . Conversely, its levels are often upregulated in Alzheimer’s disease and schizophrenia. Mirna.7z
MicroRNA-7 is a highly conserved, non-coding RNA molecule approximately 22 nucleotides long. In humans, the mature miR-7 sequence is generated from three distinct genomic loci: (Chromosome 9), MIR7-2 (Chromosome 15), and MIR7-3 (Chromosome 19). It is primarily recognized as a "network stabilizer" that maintains cellular homeostasis under environmental stress. Biological Functions and Regulation The dysregulation of miR-7 is a hallmark of
: In the pancreas, it is the most abundant endocrine miRNA in islets, where it acts as a "brake" on adult beta-cell proliferation and helps regulate insulin secretion. In humans, the mature miR-7 sequence is generated
miR-7 is preferentially expressed in neuroendocrine tissues, specifically the and pancreas .
: Its levels are controlled post-transcriptionally by "sponges" like circular RNA ciRS-7 (also known as CDR1as), which contains over 70 binding sites for miR-7 and can effectively quench its activity. Role in Pathophysiology
: Recent studies highlight its role in regulating immune responses, including T-cell activation and neuroinflammation. Clinical Potential Due to its broad regulatory reach, miR-7 is a target for:
